TIPS: 7 disturbing fact why sugar is bad for you
Added sugar is the single worst ingredient in the modern diet.
It can have harmful effects on metabolism and contribute to all sorts of diseases.
Here are (7) disturbing reasons why you should avoid added sugar like the plague.
1. Added Sugar Contains No Essential Nutrients and is Bad For Your Teeth
You’ve probably heard this a million times before… but it’s worth repeating. Added sugars (like sucrose and high fructose corn syrup) contain a whole bunch of calories with NO essential nutrients. For this reason, they are called “empty” calories.
There are no proteins, essential fats, vitamins or minerals in sugar… just pure energy. When people eat up to 10-20% of calories as sugar (or more), this can become a major problem and contribute to nutrient deficiencies.
Sugar is also very bad for the teeth, because it provides easily digestible energy for the bad bacteria in the mouth (1 ).
Bottom Line: Sugar contains a lot of calories, with no essential nutrients. It also causes tooth decay by feeding the harmful bacteria in the mouth.
2. Added Sugar is High in Fructose, Which Can Overload Your Liver
In order to understand what is so bad about sugar, then you need to understand what it is made of. Before sugar enters the bloodstream from the digestive tract, it is broken down into two simple sugars… glucose and fructose. Glucose is found in every living cell on the planet. If we don’t get it from the diet, our bodies produce it. Fructose is different. Our bodies do not produce it in any significant amount and there is no physiological need for it. The thing with fructose is that it can only be metabolized by the liver in any significant amounts. This is not a problem if we eat a little bit (such as from fruit) or we just finished an exercise session. In this case, the fructose will be turned into glycogen and stored in the liver until we need it ( 3 ).
However, if the liver is full of glycogen (much more common), eating a lot of fructose overloads the liver, forcing it to turn the fructose into fat (4 ). When repeatedly eating large amounts of sugar, this process can lead to fatty liver and all sorts of serious problems ( 5 ). Keep in mind that all of this does NOT apply to fruit. It is almost impossible to overeat fructose by eating fruit. There is also massive individual variability here. People who are healthy and active can tolerate more sugar than people who are inactive and eat a Western, high-carb, high-calorie diet.
Bottom Line: For people who are inactive and eat a Western diet, large amounts of fructose from added sugars get turned into fat in the liver.
3. Overloading The Liver With Fructose Can Cause Non- Alcoholic Fatty Liver Disease.
When fructose get turned into fat in the liver, it is shipped out as VLDL cholesterol particles. However, not all of the fat gets out, some of it can lodge in the liver.
This can lead to Non-Alcoholic Fatty Liver Disease (NAFLD), a growing problem in Western countries that is strongly associated with metabolic diseases (6 ). Studies show that individuals with fatty liver consume up to 2-3 times as much fructose as the average person (7 , 8 ).
Bottom Line: Excess fructose gets turned into fat, which can lodge in the liver and cause non-alcoholic fatty liver disease.
4. Sugar Can Cause Insulin Resistance, a Stepping Stone Towards Metabolic Syndrome and Diabetes
Insulin is a very important hormone in the body. It allows glucose (blood sugar) to enter cells from the bloodstream and tells the cells to start burning glucose instead of fat.
Having too much glucose in the blood is highly toxic and one of the reasons for complications of diabetes, like blindness. One feature of the metabolic dysfunction that is caused by the Western diet, is that insulin stops working as it should. The cells become “resistant” to it. This is also known as insulin resistance , which is believed to be a leading driver of many diseases… including metabolic syndrome, obesity, cardiovascular disease and especi
TIPS: 7 disturbing fact why sugar is bad for you
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October 27, 2016
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